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Lactating singles

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Lactating singles

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So why on earth did I put myself on stage only six months after having a child and two years of not dancing at all?

I was commissioned to create three pieces based on the experiences of undocumented students in the local community college.

Not wanting the opportunity to pass me by, I said yes, figuring I could find a handful of dancers to work with me and I would not have to be on stage.

With this project, I was hopeful for the opportunity to work only as a choreographer, someone who creates dances, rather than as a dancer who would perform the work on stage.

I needed three dancers, preferably Latinx. I found one dancer, however, the director who commissioned the work wanted an ensemble on stage.

I felt pressure to find at least a trio of dancers. At the time, he was living in Corona, CA and was commuting 16 hours round trip to Tucson every other weekend.

As such, he could not and would not commit to attending rehearsals when he was in town. I was so desperate for bodies, I put a visual artist on stage.

I felt I had no other choice: I needed to perform. Oh, a foreshadow of things to come…. The rehearsals, combined with work and mothering a nursing newborn, took a toll on my body.

I came down with lactation mastitis, which is so incredibly painful, three times while breastfeeding. Feeding times often fell during class and rehearsals, I could feel myself leaking.

The milk ducts were full, then they clogged, and I got the very painful infection. I felt like I had to suck it up and deal with it. Deal with the pain and the infections.

As an adjunct, I had little to no agency over my workload or teaching schedule. As far as the university was concerned, I was to be grateful that I had work at the institution, especially given that I do not have a PhD.

My schedule was at the mercy of sabbaticals and professors buying out classes. Oftentimes, I was notified two to three days before classes started if I was going to have a job that semester.

It was nerve-racking and stress inducing. My financial situation was always in flux because I was never sure if I would have a contract the following semester.

To make matters worse, I knew I was expendable because the one time that I tried to negotiate a contract for a bit more money, the university ceased negotiations and gave the contract to a recently graduated masters student.

Nonetheless, I accepted the offer to teach classes fall , six weeks after my child was born. I took the contract because I loved teaching and I needed the income.

When the milk supply is more firmly established, autocrine or local control system begins. During this stage, the more that milk is removed from the breasts, the more the breast will produce milk.

Low supply can often be traced to:. This is the mechanism by which milk is transported from the breast alveoli to the nipple.

Suckling by the baby stimulates the paraventricular nuclei and supraoptic nucleus in the hypothalamus , which signals to the posterior pituitary gland to produce oxytocin.

Oxytocin stimulates contraction of the myoepithelial cells surrounding the alveoli, which already hold milk.

The increased pressure causes milk to flow through the duct system and be released through the nipple. This response can be conditioned e. Milk ejection is initiated in the mother's breast by the act of suckling by the baby.

The milk ejection reflex also called let-down reflex is not always consistent, especially at first. Once a woman is conditioned to nursing, let-down can be triggered by a variety of stimuli, including the sound of any baby.

Even thinking about breastfeeding can stimulate this reflex, causing unwanted leakage, or both breasts may give out milk when an infant is feeding from one breast.

However, this and other problems often settle after two weeks of feeding. Stress or anxiety can cause difficulties with breastfeeding.

The release of the hormone oxytocin leads to the milk ejection or let-down reflex. Oxytocin stimulates the muscles surrounding the breast to squeeze out the milk.

Breastfeeding mothers describe the sensation differently. A poor milk ejection reflex can be due to sore or cracked nipples, separation from the infant, a history of breast surgery , or tissue damage from prior breast trauma.

If a mother has trouble breastfeeding, different methods of assisting the milk ejection reflex may help. These include feeding in a familiar and comfortable location, massage of the breast or back, or warming the breast with a cloth or shower.

Suckling by the baby innervates slowly-adapting [10] and rapidly-adapting [11] mechanoreceptors that are densely packed around the areolar region.

The electrical impulse follows the spinothalamic tract , which begins by innervation of fourth intercostal nerves. The electrical impulse then ascends the posterolateral tract for one or two vertebral levels and synapses with second-order neurons, called tract cells, in the posterior dorsal horn.

The tract cells then decussate via the anterior white commissure to the anterolateral corner and ascend to the supraoptic nucleus and paraventricular nucleus in the hypothalamus , where they synapse with oxytocinergic third-order neurons.

The somas of these neurons are located in the hypothalamus, but their axon and axon terminals are located in the infundibulum and pars nervosa of the posterior pituitary , respectively.

The oxytocin is produced in the neuron's soma in the supraoptic and paraventricular nuclei, and is then transported down the infundibulum via the hypothalamo-neurohypophyseal tract with the help of the carrier protein, neurophysin I , to the pars nervosa of the posterior pituitary, and then stored in Herring bodies , where they are stored until the synapse between second- and third-order neurons.

Following the electrical impulse, oxytocin is released into the bloodstream. Through the bloodstream, oxytocin makes its way to myoepithelial cells , which lie between the extracellular matrix and luminal epithelial cells that also make up the alveoli in breast tissue.

When oxytocin binds to the myoepithelial cells, the cells contract. The increased intra-aveolar pressure forces milk into the lactiferous sinuses, into the lactiferous ducts a study found that lactiferous sinuses may not exist.

A surge of oxytocin also causes the uterus to contract. During breastfeeding, mothers may feel these contractions as afterpains.

These may range from period-like cramps to strong labour-like contractions and can be more severe with second and subsequent babies. In humans, induced lactation and relactation have been observed frequently in some cultures, and demonstrated with varying success in adoptive mothers.

It appears plausible that the possibility of lactation in women or females of other species who are not biological mothers does confer an evolutionary advantage, especially in groups with high maternal mortality and tight social bonds.

Lactation can be induced in humans by a combination of physical and psychological stimulation, by drugs, or by a combination of those methods.

Rare accounts of male lactation as distinct from galactorrhea exist in historical medical and anthropological literature, although the phenomenon has not been confirmed by more recent literature.

Domperidone is a drug that can induce lactation. Charles Darwin recognized that mammary glands seemed to have developed specifically from cutaneous glands, and hypothesized that they evolved from glands in brood pouches of fish, where they would provide nourishment for eggs.

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